To Dan, I guess.
For people who are not familiar with PR, I think it might be a good idea to have an intro re:
what PR is all about for people who just drop-in-here from who-knows-where. It might also include brief descriptions of the various threads, so newbies like me can figure out where to post what. I would also like to know what the preferred method of communicating here is – as in do you like to have copies of complete research papers published, (so as to have-everything-in-the- same place) or would you like precis and references?
I took a stab at it last fall. Since then nothing much has changed. But I believe Dr. Fry is acting reasonably and in good faith. PR is not only a threat to all those that profit from the MS status quo; it is a threat to all those “diseases” like ALS, Lyme, Lupus, and all those other maladies that doctors can’t quite get a handle on. Inflammation, in some form, is the precursor of most diseases I can think of.
Some other things I have been thinking:
Does this group have an objective, other than trading information?
The reason I ask is that whatever Dr. Fry is doing could probably be made easier and faster with the application of money. Given the Internet, and the ability to network the world, we should be able to raise at least some money through crowd-funding. At this stage all this is blue-skying of course, but what-the-heck, why not? Just thinkin’…… I am a CPA, so I should be able to do something to help.
As we speak, I would guess that Big Pharma is preparing an action plan to frustrate Dr. Fry in whatever ways they can. Billions of dollars can buy a lot of frustration.
Anyhow, to start with, I have sent you a copy of what I wrote last fall. Feel free to make comments, suggestions, whatever. Note that I have had PPMS for the last 17 years, but I am fortunate enough to have maintained most of my motor functions. Unfortunately, I am taking both Percocets and morphine for my chronic – excruciating – pain. Exercise has kept me relatively limber and strong, but there is nothing to do for my pain anymore. Doctors actually look kind of sad when they tell you that.
September 30, 2012
To whom it may concern:
MS ………………….. and, “PROTOMYXZOA RHEUMATICA”
I believe that the MS community should be aware of something that might change how we all look at disease, particularly how we look at disease blamed on a dysfunctional immune system.
This is because an American researcher has shown that MS is caused by a newly discovered protozoan pathogen that is able to escape being detected by our immune system. CCSVI is an aggravating symptom, not a primary cause.
In November of 2011, Marc Braman, MD,MPH, interviewed Steven Fry, MD, MSc,. regarding research being done by Dr. Fry. on the origins of chronic inflammatory disease in general, and on the origins of neurodegenerative disease in particular. What follows is my interpretation of information contained in a transcript of this interview. The actual transcript may be found at http://www.iadvocatehealth.org/protozoal_infection0.aspx
Please note that the research mentioned in this interview is still very much in the exploratory stage, and Dr. Fry has made no claims to have cured anybody of anything. That being said, Dr. Fry is doing fascinating work on the frontiers of disease etiology, and has made and documented some truly remarkable results.
BACKGROUND
About twenty years ago an American doctor by the name of Steven Fry became intrigued by the notion that chronic inflammatory diseases like rheumatism could be treated by antibiotics. As an MD in general practice with a master’s degree in molecular biology and experience in surgical pathology, Dr. Fry had a history of experience and training which gave him a unique perspective when it came to developing possible models of disease.
But most of all he had patients with symptoms, and the curiosity to look wherever the science led him when it came to treating these symptoms. Dr. Fry is an actual research professional, and as such is unlike the majority of neurologists who start their research only after thorough indoctrination in the dysfunctional autoimmune model of MS.
So, about ten years ago Dr. Fry said “…..I just borrowed money, bought a nice research grade microscope, and started developing some stains”. Thus began his Odyssey in search of what causes MS, ALS, chronic fatigue syndrome, fibromyalgia, diabetes 1, diabetes 2, Parkinson’s, lupus, rheumatism, Alzheimer’s, etc. …. and of course CCSVI.
WHAT DR. FRY FOUND – “PROTOMYXZOA RHEUMATICA”
After looking, and looking, and looking …………. and then honing the sophisticated research techniques needed to see and record what no other researcher had ever seen and recorded ………. Dr. Fry was able to tease out signs of a new, never seen before, “critter” in the blood of some of his patients with chronic neurodegenerative disease. It was a kind of a protozoa, a blood-loving parasite very close to malaria.
Dr. Fry has named this “critter” protomyxzoa rheumatica”.
One of its distinguishing features was that rather than float or swim in the blood, this organism liked to form biofilm communities. A biofilm is an aggregate of microorganisms in which cells adhere to each other, especially on a surface.
In the case of protomyxzoa rheumatica, biofilms can form on the walls of blood vessels, and tends to act as a coagulating substance within the blood that resembles normal clotting behaviour, but is not normal clotting behaviour. In a biofilm a dense extracellular matrix and an outer layer of cells can protect the interior of a biofilm community, in some cases increasing antibiotic resistance a thousandfold.
Upon sequencing the genome, Dr. Fry was able to unequivocally say that this organism he discovered is indeed unique. He can grow it in the lab.
HIDING IN PLAIN SIGHT – THE STEALTH DISEASE
So, if protomyxzoa rheumatica is a disease pathogen, why doesn’t our immune system detect it and destroy it like it does other pathogens, and why don’t some people who have protomyxzoa rheumatica ever seem to get a disease? Good questions.
Protomyxzoa rheumatica has evolved along with the rest of us over thousands of years. Early on this micro-organism found it could improve its chances of survival by incorporating some of its host’s genes into its own physiology. It has managed to do this enough times to finally look enough like regular human tissue so as to be ignored by the body’s immune system.
Virtually everyone seems to have protomyxzoa rheumatica – we all age, and the older we are, the more protomyxzoa rheumatica we have in our blood. So no one is completely immune from protomyxzoa rheumatica – some of us just manage to stay healthier than others.
Protomyxzoa rheumatica (which probably comes in different strains) may manifest itself in many ways depending on one’s genetic predisposition and environment. So one person will get one thing (eg. MS) and someone else will get another (eg. lupus), and some people will just continue to get worsening circulation (greater systemic infection) until they die.
Protomyxzoa rheumatica is a very complex organism. Because of its inclusion in a biofilm, and its slow rate of growth, it’s really not very invasive. Unlike staph, strep, or malaria for example, it can often hide itself completely from the immune system – the ultimate “stealth fighter”, so to speak.
PROTOMYXZOA RHEUMATICA – AND CCSVI
By now you have probably guessed the tie-in between protomyxzoa rheumatica and CCSVI. With protomyxzoa rheumatica, biofilm “sludge” tends to accumulate on the walls of blood vessels, and is particularly noticeable in veins such as those that drain the central nervous system back to the heart.
Protomyxzoa rheumatica can just cause CCSVI; it can cause CCSVI followed by MS; or protomyxzoa rheumatica can cause MS all on its own. This helps to explain some of the inconsistencies found while researching CCSVI.
Having attended at Buffalo, Dr. Fry is familiar with the work of Dr. Zamboni, and aware of research done by Dr. Hubbard. Dr. Fry is even able to observe “vegetations” (coagulating biofilm) in venous ultrasounds of the type Dr. Zamboni uses.
And Dr. Fry thinks it is quite logical for some people to experience dramatic improvements following CCSVI Treatment in which these blockages of vegetation are removed, immediately improving blood flow. The problem of course is that the “vegetation” simply regrows, causing restenosis.
In hindsight, it is easy to see how researchers could get confusing results when they believed they were working with the same data. It is still interesting to note, however, the bias that seemingly crept into supposedly “scientific” observation depending on the extent of CCSVI “faith” held by the observer.
PROTOMYXZOA RHEUMATICA – VITAMIN D & HEAT
It has long been noted that people who live in the tropics seldom get MS. These people get plenty of sunshine which in turn produces Vitamin D, and of course warmer weather. Dr. Fry has noted that protomyxzoa rheumatica hates heat, and that his patients need Vitamin D supplements before they can get better.
PROTOMYXZOA RHEUMATICA – GENERAL DIET
Dr. Swank is famous among MS patients for his low-fat diet, which has proven to have helped many MS sufferers. Well, guess what? Dr. Fry has found that protomyxzoa rheumatica not only thrives on fat – it positively seems to need it in order to exist. No fat, no protomyxzoa rheumatica – at least not very much.
Dr Fry has suggested that many people with chronic illness may struggle with wheat not due to the gluten content, but due to the high levels of an amino acid called arginine, which the protomyxzoa rheumatica protozoan thrives on.
PROTOMYXZOA RHEUMATICA – TICKS & MOSQUITOS
Lyme disease and MS have often been confused with each other, which is perfectly natural considering they are probably caused by the same thing – protomyxzoa rheumatica. Dr. Fry believes ticks are a vector for protomyxzoa rheumatica, as are mosquitos.
Since mosquitos are found almost anywhere, it is easy to see how protomyxzoa rheumatica can get spread from person to person and generation to generation. People get it when they are young, and as they age it gets ever more prevalent in the blood, causing ever more disease.
PROTOMYXZOA RHEUMATICA – THE BAD NEWS
Protomyxzoa rheumatica has not only been able to mostly hide from our immune system, it has been able to mostly hide from all the antibiotics doctors have been giving us to fight other diseases. Current antibiotics may sometimes weaken protomyxzoa rheumatica, but they cannot eradicate it.
There are two issues to be dealt with:
1. There is a need to find an antibiotic or cocktail of antibiotics available in therapeutic doses that will kill protomyxzoa rheumatica; and
2. There is a need to find a way to safely overcome something called the Jarisch-Herxheimer reaction, a clinical worsening due to an infectious agent dying off. Basically what happens is that once antibiotics begin to kill protomyxzoa rheumatica, a person’s own immune system “wakes up” to find the body with massive blood poisoning, says OMG, and responds with a massive inflammatory response that is deadly.
As Dr. Fry says, he could come up with the ideal antibiotic to kill protomyxzoa rheumatica tomorrow, but couldn’t use it for fear of killing his patient.
It is important to note that many actions taken to decrease protomyxzoa rheumatica, even dieting, may induce a Jarisch-Herxheimer type response. Unfortunately, treating protomyxzoa rheumatica effectively often produces a situation in which “You are going to feel worse before you get better”.
So the challenge is to find an effective treatment for protomyxzoa rheumatica, and then to find a way to safely administer it.
PROTOMYXZOA RHEUMATICA – THE GOOD NEWS
The problem of having any kind of “silver bullet” is that you have to know what the target is before you shoot. Well, now we know the target. Protomyxzoa rheumatica.
Thanks to the Internet, research can be speeded up by sharing information. Powerful computers are now available to crunch numbers when this is required, as in sequencing a genome.
PROTOMYXZOA RHEUMATICA – NOT JUST MS
There is a lot of research that points to the fact that many diseases, not just MS, stem from some form of chronic inflammation. If protomyxzoa rheumatica turns out to be the “premier pathogen”, much of the world of medicine is going to be turned upside down. There are a lot of very powerful people in very powerful places that are going to get very upset.
People with MS can now find common cause with many other people who are being treated as if a symptom was really a disease. ALS, chronic fatigue, fibromyalgia, diabetes 1, diabetes 2, Alzheimers, lupus ….. the list goes on and on. Dr. Fry does not mention it, but there is considerable evidence that it is chronic inflammation that causes cancer.
There is strength in numbers. There is reason to believe that the assembled might of patients who find they are all fighting the same battle will overcome the obstacles placed in their path. There is hope.
WHAT NOW FOR CCSVI?
The fact that CCSVI may not be a primary cause of MS does nothing to change the fact that many MS patients who have CCSVI will get better once their CCSVI is treated. For this reason alone, CCSVI Treatment for people with severe and/or end stage MS should be made available immediately. To not do so is unconscionable. But of course that cannot bother someone without a conscience.
The upside risk of CCSVI is huge. The downside risk is tiny. Let it be.
But I really don’t believe that the trials in B.C. will change anything. “Inconclusive” is my best guess, somewhat like Buffalo. Meanwhile MS patients will continue to suffer and die.
THE COMING WAR
Dr. Fry has not as yet engaged the patient public at large. It was mostly luck that I happened to stumble on to a transcription of the interview Dr. Fry did with Dr. Braman.
I have made contact with Dr. Fry’s office, and with Dr. Fry’s lawyer. They are a very impressive group of people, but they face a mighty challenge. And they know it. So far, Dr. Fry has submitted papers to four neurological journals. They have all turned him down. Where have we seen that before!
If I am correct, there is about to be a sea change in medicine that we have not seen in decades, a definite “disruptive technology” that is going to change medical history.
(“Disruptive technology” is a term coined by Harvard Business School professor Clayton M. Christensen to describe a new technology that unexpectedly displaces an established technology.)
In his 1997 best-selling book, “The Innovator’s Dilemma,” Christensen separates new technology into two categories: sustaining and disruptive. Sustaining technology relies on incremental improvements to an already established technology. Disruptive technology drives old technology right out of the marketplace. No more films needed for cameras – Kodak just went broke. Many doctors are going to have to find something else to do.
Big Pharma is totally at risk. Whole medical disciplines are threatened. No more Pftzer. No more rheumatologists. And certainly less neurologists.
There is going to be war.
IT IS IMPORTANT TO NOTE AFTER READING THE ABOVE THAT CCSVI TREATMENT IS JUST AS IMPORTANT AS EVER! CCSVI TREATMENT DOES OFTEN STOP THE SYMPTOMS OF MS, BUT SOMETIMES ONLY FOR A LITTLE WHILE. THAT IS BECAUSE THE “GERM” THAT CAUSES CCSVI IS STILL ALIVE IN THE BLOOD, AND WILL OFTEN CAUSE RESTENOSIS.
WHILE CCSVI MAY TRIGGER MS, IT IS PROTOMYXZOA RHEUMATICA THAT HAS AIMED THE GUN

• This topic was modified 10 years, 1 month ago by jasonyohon.